DISEASE: Alfalfa dwarf
HOST: Alfalfa
Alfalfa dwarf is characterized by stunted plants (left), usually dark blue-green in color. Xylem tissues become brown, especially when exposed to air.
Alfalfa dwarf | Alfalfa
DISEASE: Alfalfa dwarf
HOST: Alfalfa (Medicago sativa)
PATHOGEN: Xylella fastidiosa
SOURCE: R. M. Davis
DISEASE: Bacterial leaf streak and stripe
HOST: Grass
Vasey grass with yellowish to brownish red streaks and stripes.
Bacterial leaf streak and stripe | Grass
DISEASE: Bacterial leaf streak and stripe
HOST: Grass (Paspalum urvillei)
PATHOGEN: Acidovorax avenae
PATHOGEN SYNONYM: Acidovorax avenae subsp. avenae
SOURCE: R. Gitaitis
DISEASE: Citrus stubborn disease
HOST: Citrus (sp. unknown)
Characteristic symptoms of thickened peel at peduncle end and aborted seeds.
Citrus stubborn disease | Citrus (sp. unknown)
DISEASE: Citrus stubborn disease
HOST: Citrus (sp. unknown) (Citrus sp.)
PATHOGEN: Spiroplasma citri
SOURCE: APS
DISEASE: Citrus stubborn disease
HOST: Citrus (Orange)
Severely stunted sweet orange tree. Foliage is dense and abnormally upright. Leaves may be cupped and unusually thick. They also may be chlorotic and mottled.
Citrus stubborn disease | Citrus (Orange)
DISEASE: Citrus stubborn disease
HOST: Citrus (Orange) (Citrus sinensis)
PATHOGEN: Spiroplasma citri
SOURCE: S. M. Garnsey
DISEASE: Citrus stubborn disease
HOST: Citrus (Orange)
Fruit from a diseased tree are frequently lopsided or acorn-shaped, usually few and small. They may not color at stem end.
Citrus stubborn disease | Citrus (Orange)
DISEASE: Citrus stubborn disease
HOST: Citrus (Orange) (Citrus sinensis)
PATHOGEN: Spiroplasma citri
SOURCE: J. M. Bove, M. Garnier
DISEASE: Citrus stubborn disease
HOST: Citrus (Orange)
Sweet orange exhibiting off-season flowering and crop heterogeneity. Diseased trees generally have shoots with shortened internodes, which lead to rosettes with cupped leaves.
Citrus stubborn disease | Citrus (Orange)
DISEASE: Citrus stubborn disease
HOST: Citrus (Orange) (Citrus sinensis)
PATHOGEN: Spiroplasma citri
SOURCE: J. M. Bove, M. Garnier