DISEASE: Bacterial foot rot
HOST: Rice
The disease causes sheaths to turn dark brown and rot; dead leaves droop. Nodes, culms, and crowns also decay, and infected tillers are easily detached from the crown. Culms and internodes turn black.

Bacterial foot rot | Rice
DISEASE: Bacterial foot rot
HOST: Rice (Oryza sativa)
PATHOGEN: Dickeya zeae
PATHOGEN SYNONYM: Erwinia chrysanthemi pv. zeae
SOURCE: M. Goto
DISEASE: Bacterial foot rot
HOST: Rice
Decayed culms (right) and healthy culms (left). Leaf sheaths of infected plants exhibit dark brown decay and attached leaves turn yellow and wilt.

Bacterial foot rot | Rice
DISEASE: Bacterial foot rot
HOST: Rice (Oryza sativa)
PATHOGEN: Dickeya zeae
PATHOGEN SYNONYM: Erwinia chrysanthemi pv. zeae
SOURCE: M. Goto
DISEASE: Oleander knot
HOST: Oleander
Oleander with small knots/galls on leaf and stems.

Oleander knot | Oleander
DISEASE: Oleander knot
HOST: Oleander (Nerium oleander)
PATHOGEN: Pseudomonas savastanoi pv. nerii
SOURCE: W. Sinclair
DISEASE: Oleander knot
HOST: Oleander
Young stem with knots/galls.

Oleander knot | Oleander
DISEASE: Oleander knot
HOST: Oleander (Nerium oleander)
PATHOGEN: Pseudomonas savastanoi pv. nerii
SOURCE: R. Raabe
DISEASE: Oleander knot
HOST: Oleander
Leaf with early stage of knot/gall formation.

Oleander knot | Oleander
DISEASE: Oleander knot
HOST: Oleander (Nerium oleander)
PATHOGEN: Pseudomonas savastanoi pv. nerii
SOURCE: R. Raabe
DISEASE: Oleander knot
HOST: Oleander
Infected flowers with small knots/galls and necroses.

Oleander knot | Oleander
DISEASE: Oleander knot
HOST: Oleander (Nerium oleander)
PATHOGEN: Pseudomonas savastanoi pv. nerii
SOURCE: W. Sinclair
DISEASE: Syringae leaf spot
HOST: Tomato
Leaves with brown necrotic lesions and chlorotic margins. Symptoms vary greatly among cultivars. Some have black or brown lesions with bright yellow, chlorotic areas and others do not have yellowing.

Syringae leaf spot | Tomato
DISEASE: Syringae leaf spot
HOST: Tomato (Lycopersicon esculentum)
PATHOGEN: Pseudomonas syringae pv. syringae
SOURCE: R. Gitaitis