DISEASE: Bacterial leaf spot
HOST: Pothos
Leaf with black necrotic tissues with concentric rings of light and dark tissues.

Bacterial leaf spot | Pothos
DISEASE: Bacterial leaf spot
HOST: Pothos (Epipremnum pinnatum)
PATHOGEN: Pseudomonas cichorii
SOURCE: APS
DISEASE: Bacterial wilt
HOST: Pothos
Pothos with infected stem and petioles.

Bacterial wilt | Pothos
DISEASE: Bacterial wilt
HOST: Pothos (Epipremnum aureum)
PATHOGEN: Ralstonia solanacearum
PATHOGEN SYNONYM: Pseudomonas solanacearum
SOURCE: D. Norman
DISEASE: Bacterial wilt
HOST: Pothos
Plant with severely infected leaves and stems.

Bacterial wilt | Pothos
DISEASE: Bacterial wilt
HOST: Pothos (Epipremnum aureum)
PATHOGEN: Ralstonia solanacearum
PATHOGEN SYNONYM: Pseudomonas solanacearum
SOURCE: D. Norman
DISEASE: Olive knot
HOST: Olive
Multiple infections of young stems. The bacterium invades vascular tissues during certain times of the year and may be isolated from branches that appear healthy.

Olive knot | Olive
DISEASE: Olive knot
HOST: Olive (Olea europaea)
PATHOGEN: Pseudomonas savastanoi pv. savastanoi
SOURCE: M. Schroth
DISEASE: Olive knot
HOST: Olive
Tree with knots/galls on branches along with twig dieback, which is associated with knots. Fusarium and Diplodia spp. infect through knots and are thought to be main reason for dieback.

Olive knot | Olive
DISEASE: Olive knot
HOST: Olive (Olea europaea)
PATHOGEN: Pseudomonas savastanoi pv. savastanoi
SOURCE: M. Schroth
DISEASE: Olive knot
HOST: Olive
Multiple infections on olive branch. Knots at this stage begin to die from the outside in and are infected by several fungi.

Olive knot | Olive
DISEASE: Olive knot
HOST: Olive (Olea europaea)
PATHOGEN: Pseudomonas savastanoi pv. savastanoi
SOURCE: M. Schroth
DISEASE: Slime flux
HOST: Casuarina
Slime flux occurs on a number of tree species and is always associated with bacteria and yeast. However, the causal agent has not been identified.

Slime flux | Casuarina
DISEASE: Slime flux
HOST: Casuarina (Casuarina equisetifolia)
PATHOGEN: Causal agent unknown
SOURCE: W. Sinclair