DISEASE: Bacterial wilt
HOST: Alfalfa
Longitudinal section of defined brown gum pockets on inner surface of root bark.
Bacterial wilt | Alfalfa
DISEASE: Bacterial wilt
HOST: Alfalfa (Medicago sativa)
PATHOGEN: Clavibacter michiganensis subsp. insidiosus
SOURCE: S. Thomson
DISEASE: Bacterial wilt
HOST: Alfalfa
Infected plant (left) with typical symptoms of stunting and chlorosis. Other characteristic symptoms are spindly stems and small distorted leaflets. Healthy plant (right).
Bacterial wilt | Alfalfa
DISEASE: Bacterial wilt
HOST: Alfalfa (Medicago sativa)
PATHOGEN: Clavibacter michiganensis subsp. insidiosus
SOURCE: S. Thomson
DISEASE: Bacterial wilt
HOST: Alfalfa
Diseased plant with chlorotic leaves.
Bacterial wilt | Alfalfa
DISEASE: Bacterial wilt
HOST: Alfalfa (Medicago sativa)
PATHOGEN: Clavibacter michiganensis subsp. insidiosus
SOURCE: S. Thomson
DISEASE: Bacterial wilt
HOST: Alfalfa
Cross sections of diseased roots with defined brown gum pockets on inner surface of bark. Healthy root (upper left). Other sections represent increases in disease severity.
Bacterial wilt | Alfalfa
DISEASE: Bacterial wilt
HOST: Alfalfa (Medicago sativa)
PATHOGEN: Clavibacter michiganensis subsp. insidiosus
SOURCE: L. Claflin
DISEASE: Slippery skin
HOST: Onion
Rot progresses from the top of infected scales and eventually internal tissues rot. In early stages, the only external symptoms may be softening of the neck.
Slippery skin | Onion
DISEASE: Slippery skin
HOST: Onion (Allium cepa)
PATHOGEN: Burkholderia gladioli pv. alliicola
PATHOGEN SYNONYM: Pseudomonas gladioli pv. alliicola
SOURCE: H. Schwartz
DISEASE: Sweet potato scab
HOST: Sweet potato
Tubers with large, dark scabs.
Sweet potato scab | Sweet potato
DISEASE: Sweet potato scab
HOST: Sweet potato (Ipomoea batatas)
PATHOGEN: Streptomyces ipomoeae
SOURCE: APS
DISEASE: Sweet potato scab
HOST: Sweet potato
Black and necrotic decay of fibrous roots.
Sweet potato scab | Sweet potato
DISEASE: Sweet potato scab
HOST: Sweet potato (Ipomoea batatas)
PATHOGEN: Streptomyces ipomoeae
SOURCE: W. Martin